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18 Coronavirus Autopsies (This is what they found in the Brain) | COVID-19 Autopsy


What Doctors Are Learning From Autopsy Findings of New CORONAVIRUS Patients

What Doctors Are Learning From Autopsy Findings of New CORONAVIRUS Patients

⏩ Timestamps, click to skip ahead!
00:00 - Common Systoms of COVID
01:40 - What we know about COVID
02:25 - Early findings of Multiple Cutopsy and Biopsy Reports of COVID
03:02 - Microscope Picture of the Coronavirus and Kidney Cells
03:20 - COVID Autopsy Findings

Once the SARS-CoV-2 virus is deeply embedded in the body, it begins to cause more severe disease. This is where the direct attack on other organs that have ACE2 receptors can occur, including heart muscle, kidneys, blood vessels, liver, and the brain. Early findings, including those from multiple autopsies and biopsy reports, show that viral particles can be found not only in the nasal passages and throat, but also in tears, stool, kidneys, liver, pancreas, and heart. One case report found evidence of viral particles in the CSF, meaning the fluid around the brain. That patient had meningitis.

So the coronavirus is sometimes going to all these different organs by means of attaching to the ACE2 receptors that are there, but that’s not even the whole story.

Because in some COVID-19 cases, by the time the body’s immune system figures out the body are being invaded, it's like unleashing the military to stomp out the virus, and in that process, there’s a ton of collateral damage. This is what we refer to as the cytokine storm. When the COVID gets into the alveolar cells, meaning the tiny little air sacs within the lungs, it makes a ton of copies of itself and goes onto invading more cells. The alveoli’s next-door neighbor is guessed who, yeah, the tiniest blood vessels in our body, capillaries. And the lining of those capillaries is called the endothelium, which also has ACE2 receptors. And once the coronavirus invades the capillaries. It means that it serves as the trigger for the onslaught of inflammation AND clotting. Early autopsy results are also showing widely scattered clots in multiple organs. In one study from the Netherlands, 1/3rd of hospitalized with COVID 19 got clots despite already being on prophylactic doses of blood thinners. So not only are you getting the inflammation with the cytokine storm, but you’re also forming blood clots, that can travel to other parts of the body, and cause major blockages, effectively damaging those organs.

So it can cause organ damage by
1) Directly attacking organs by their ACE2 receptor - Yes!
2) Indirectly attacking organs by way of collateral damage from the cytokine storm - Yes!
3) Indirectly cause damage to organs by means of blood clots - Yes!
4) Indirectly cause damage as a result of low oxygen levels, improper ventilator settings, drug treatments themselves, and/or all of these things combined - Yes!

Endothelial cells are more vulnerable to dying in people with preexisting endothelial dysfunction, which is more often associated with being a male, being a smoker, having high blood pressure, diabetes, and obesity. Blood clots can form and/or travel to other parts of the body. When blood clots travel to the toes, and cause blockages in blood flow there, meaning ischemia or infarction, that can cause gangrene there. And lots of times patients with gangrene require amputation, and “COVID toes”

So is antiphospholipid antibody syndrome, the cause of all these blood clots in patients with severe COVID? Maybe. Some patients with APS have what’s called catastrophic APS, where these patients can have strokes, seizures, heart attacks, kidney failure, ARDS, skin changes like the ones I mentioned. Viral infectious diseases, particularly those of the respiratory tract, have been reported as being the triggers for CAPS.

Various factors increase the risk of developing arterial thrombosis. Classically, the cardiovascular-dependent risk factors implicated in clotting have been hypertension, meaning high blood pressure, high levels of cholesterol, smoking, diabetes, age, chemotherapy, and degree of infection. All of these contribute toward developing arterial thrombosis.

A lot of patients with severe COVID 19 have certain labs that resemble DIC, such as increased PT/INR, increased PTT, decreased levels of platelets. But the reason why these Coronavirus patients who developed clots in the study I mentioned earlier, the reason why they don’t have DIC, is actually 2 reasons, one, they weren’t having extensive bleeding, and two, they did not have low fibrinogen levels. And if its truly DIC, you would have both of those things.

Anyway, you can probably glean from this video why it's so hard for doctors to figure out what is going on with this virus. Between the variable ways this disease can present in different patients, and the different ways that organs can suffer damage, yeah, this is really, really really, complicated.

Are BLOOD CLOTS the reason why COVID-19 patients are dying?
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Dr. Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine

Coronavirus Autopsy Report Analysis by Dr. Mike | COVID-19 Autopsy

Coronavirus Autopsy Report Analysis by Dr. Mike | COVID-19 Autopsy
#coronavirus #covid19 #covid_19

⏩ Timestamps, click to skip ahead!
00:00 - Introduction
01:00 - Current Positive COVID Cases Details
02:05 - New Studies of COVID and Autopsy Report Analysis of a COVID Patient

Coronavirus | COVID-19 YouTube Video Playlist:

Coronavirus is the virus responsible for the COVID-19 outbreak. Wuhan, China has been the epicenter of this epidemic, but some experts, like Dr. Anthony Fauci, are now saying that we are on the verge of a pandemic.

Before I get to the Coronavirus autopsy report of a patient with COVID-19, its important to understand the context of the numbers of total people infected, total people with coronavirus pneumonia, number of people who developed ARDS, and the total number of deaths.

When looking at the numbers, we should realize that they are almost certainly being underreported in China, and there are multiple reasons for that, which I won't get into right now.

Although these are not concrete numbers, its what we have to go by at this point. The percentage of people.

Also, up to this point, there has not been any pathology reported on this disease because of limited access to autopsy and biopsy reports.

But finally, we now have a new case report study in Lancet Respir Med, published Feb 17, that has autopsy reports for a patient who died from COVID-19.

Pathological findings of COVID-19 associated with acute respiratory distress syndrome.

A patient is a 50-year-old man from China, who visited Wuhan Jan 8–12. On Jan 14, he developed a dry cough and some mild chills, so this is day 1 of the illness. However, he did not initially seek medical attention and kept working until Jan 21. He then went to a medical clinic on Jan 21, because by that time, he had developed worsening symptoms. He had fever, chills, fatigue, cough, and shortness of breath.
On Jan 22 (day 9 of illness), the Beijing Centers for Disease Control (CDC) confirmed by reverse real-time PCR assay that the patient had COVID-19.

He was immediately admitted to the isolation ward and received supplemental oxygen through a face mask.

He was given several different medications, which included the inhaled version of interferon alfa-2b, lopinavir plus ritonavir as antiviral therapy, and Moxifloxacin, to prevent secondary bacterial infection.

He was also given a steroid, methylprednisolone, to attenuate lung inflammation.

On day 12 of illness, after the initial presentation, his symptoms did not improve, other than his fever, which he received medication for.

His chest x-ray on day 12 showed progressive bilateral infiltrates. He repeatedly refused ventilator support in the intensive care unit repeatedly, apparently because he suffered from claustrophobia.

His oxygen saturation values decreased to 60%, and the patient had a cardiac arrest. At that point he was intubated with mechanical ventilation, he had chest compressions and epinephrine.

Unfortunately, they are unable to revive him.

An autopsy is done, and biopsy samples were taken from the lung, liver, and heart.

The heart tissue was essentially normal.

The liver biopsy of this patient showed moderate microvascular steatosis and
mild lobular and portal activity, indicating the injury could have been caused by either Coronavirus infection or as a result drug-induced liver injury.

Histological examination of lung tissue showed diffuse alveolar damage with cellular fibromyxoid exudates, along with the desquamation of pneumocytes and hyaline membrane formation.

These findings are consistent with acute respiratory distress syndrome ( ARDS ).

Interstitial mononuclear inflammatory infiltrates, dominated by lymphocytes, was seen in both lungs. There were multinucleated syncytial cells with atypical large alveoli characterized with prominent nucleoli, consistent with viral cytopathic-like changes.

These pathological features of COVID-19 greatly resemble those seen in SARS and Middle Eastern respiratory syndrome (MERS) coronavirus infection.

Acute Respiratory Distress Syndrome ( ARDS )
To watch the video please visit this link:

Dr. Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine

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#coronavirus #covid19 #covid_19

188 Coronavirus Autopsies (COVID-19) – Possible Heart Inflammation (Myocarditis)?

Coronavirus Autopsy Report | 188 Coronavirus Autopsies (COVID-19) – Possible Heart Inflammation (Myocarditis)?

One-third of Big Ten athletes who contracted COVID-19 show signs of heart inflammation. Is SARS-CoV-2 really causing this?

Coronavirus | COVID-19 YouTube Video Playlist:

This video is a summary of all the coronavirus autopsy findings that have been done on coronavirus patients. This is based on 8 published studies and/or case reports. Here are the links to those studies:

SARS-CoV-2 exhibits selectivity for the lungs. Specifically, type II pneumocytes, meaning type II alveolar cells. Alveoli are the tiny microscopic air sacs of the lungs, which is the part of our lungs that is responsible for gas exchange. Air is brought down into the lungs, to the alveoli, and the oxygen diffuses from the alveoli into our tiny blood vessels there, called capillaries. At the same time, carbon dioxide, a waste product from our body, travels from the capillaries into our alveoli, and we then exhale out that carbon dioxide. Alveoli are made up of mainly type I alveolar cells. But to a lesser degree, they are also made up of type II alveolar cells, and these guys are sort of like the maintenance guys for the alveoli. They play a part in making surfactant, a sort of lubricant for the alveoli. But these cells also play a part in defending against foreign pathogens, like viruses and bacteria.

Well as it turns out these type II alveolar cells have the ACE2 receptors on them, and SARS-CoV-2 binds to this receptor, and that’s how it gains entry into these cells, and into our body. When the SARS-CoV-2 invades the type II alveolar cells, it precipitates a cascade of reactions that causes the body to react to it, with inflammation, and lots of damage to the alveoli, known as diffuse alveolar damage. Clinically, this is what we call ARDS, acute respiratory distress syndrome. This is what causes oxygen levels to go down, and what causes the so-called cytokine storm. When people die of COVID, this is what’s going. Also, there is a propensity for blood clots to develop, and some people with COVID died as a result of pulmonary emboli, meaning blood clots in their lungs. The capillaries in the lung surround the alveoli. Here, they serve to bring red blood cells in close proximity to the alveoli, to allow gas exchange to occur, as I mentioned earlier. The lining of these capillaries is called the endothelium, the cells that make up the endothelium here, also have ACE2 receptors. The virus, at least in those with severe disease, seems to be infiltrating the endothelium and causing inflammation and injury to the capillaries, not just the alveoli. This likely at least partially explains why this virus is causing blood clots to develop here.

So we are seeing a common theme here, and that is microthrombi that are being found in blood vessels of pretty much all the organs, including brain, kidneys, heart, liver, and of course lungs. This is likely all because of endothelial damage that occurs as a result of the virus binding to the ACE2 receptors that are located there.

After all, in some of these coronavirus autopsy studies, they used electron microscopy to find what appeared to be viral particles in the endothelial cells not only in the lungs, but also in the heart and kidneys.The endothelial damage serves to trigger the clotting process, something known as a coagulation cascade. But its also possible that the endothelial damage is mainly occurring in the lung capillaries, and that’s where the tiny clots first develop, and then they end up traveling to other parts of the body, eventually lodging in blood vessels of other organs. Or it could be both of these things. It's interesting to note that Endothelial cells are more vulnerable to dying in people with preexisting endothelial dysfunction, which is more often associated with being a male, being a smoker, having high blood pressure, diabetes, and obesity. So overall, organ damage that occurs in severe COVID- is likely a result of a multitude of factors.

Dr. Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
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#coronavirus #covid19 #covid_19

Exclusive interview with forensic expert: First COVID-19 autopsy provides critical insight

China conducted the first autopsy on a patient who died from novel coronavirus pneumonia (COVID-19), the pathology report for which was released on Friday. What important information does the autopsy reveal? CCTV conducted an exclusive interview with Professor Liu Liang who performed the first COVID-19 autopsy. #COVID19 #coronavirus #autopsy

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Doctors warn of potentially deadly blood clots in COVID-19 patients

Doctors are warning about the mysterious and dangerous problem of blood clotting among some coronavirus patients. CBS News senior medical correspondent Dr. Tara Narula explores the new symptom and a potential new treatment that can help.

ICU Doctor: Top 10 Things I learned Treating Coronavirus Patients | Coronavirus Intensive Care

ICU Doctor: Top 10 Things I learned Treating Coronavirus Patients | Coronavirus Intensive Care
#coronavirus #covid19 #covid_19

Coronavirus | COVID-19 YouTube Video Playlist:

Some intensive care units in various hospitals throughout this county have designated units for COVID-19 patients. As an intensive care doctor, I’ve been seeing a lot of COVID-19 patients in our designated Coronavirus ICU. It's one thing to read about Coronavirus in the medical literature, but to be actually seeing real patients with this disease is another experience altogether. There are a lot of things I’ve learned, but for this video, I’ll focus on the top 10 that stand out to me.

So, starting at the bottom with number 10, is, signs/symptoms. The most common symptoms that I’m seeing are fever, cough, shortness of breaths, and body/muscle aches. I haven’t seen many patients with the other symptoms we often hear about, such as loss of taste and smell, or nausea, and diarrhea. I have not seen any rashes related to Coronavirus, probably because I only see adult patients. I will say that even though confusion and delirium are very common in the ICU in general, there does seem to be more of that with Coronavirus.

A lot of COVID-19 patients who require hospitalization have low levels of vitamin D. And this is consistent with what we are seeing in a lot of recent studies that have been coming out. But of course, correlation doesn’t necessarily mean causation, so does it just so happen that a lot of patients who have moderate or severe COVID, happen to have low vitamin D levels? Maybe, maybe not. And does that mean that we should give every hospitalized patient with Coronavirus big doses of vitamin D when they hit the door? Maybe. And does that mean people, in general, should supplement with vitamin D? And what is the ideal level of vitamin D for the population, especially when it comes to COVID-19? Should we be targeting the current general recommendation for everyone, irrespective of COVID-19, with a goal of 20 ng/ml? or should we aim for higher, like 30, or perhaps 40? No one knows for sure the answers to these questions. But there are studies being done on this. And as we speak there are 3 RCT for vitamin D and Coronavirus.

This virus is VERY contagious. One of my patients was in the hospital for unrelated reasons. She actually had sepsis due to infarcted gut, meaning part of her intestine was not getting enough blood flow. It was severe enough to the point that some of the tissue in her intestine had died. When this happens, the bacteria that live in the intestine can then invade the walls of the intestine, and get into the bloodstream. This is bad news because these bacteria can then spread throughout the body, and this is known as sepsis. Besides antibiotics, this treated with surgery, where the dead gut tissue is removed, meaning part of the intestine is taken out. And this is what happened to her. And she got better. But after she initially got better, she started having more difficulty with her breathing. Her oxygen levels were dropping, despite us giving her more and more oxygen. So we got a CXR, and later a CT scan of the chest, which showed bilateral infiltrates, meaning areas of inflammation in both lungs. And this is the pattern we typically see with COVID-19 pneumonia, where it tends to go to the periphery of the lungs and also more so at the bottom of the lungs.

Vitamin D3 (Cholecalciferol) and Vitamin D2 (Ergocalciferol) and Calcitriol
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Does Vitamin D help with Immunity?
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Dr. Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
Instagram Account:

#coronavirus #covid19 #covid_19

Coronavirus Pandemic Update 75: COVID-19 Lung Autopsies - New Data

COVID-19 Update 75 with Roger Seheult, MD. All coronavirus updates available free at our website

A new article from the New England Journal of Medicine details observations of endothelial damage and thrombosis in lung autopsies of patients who had COVID-19. This report supports the hypothesis that Dr. Seheult has illustrated over the past few weeks.

Dr. Seheult also discusses the re-opening of the largest county in California and reviews a recent article published in Nature about how T-cells found in COVID-19 patients is a favorable sign for long-term immunity. (This video was recorded on May 25, 2020)

Links referenced in this video:

Johns Hopkins Tracker -


Washington Post -

Science -


National Geographic -

Bronchoscopy -


Some previous videos from this series (visit for the full series):
- Coronavirus Pandemic Update 74: Vitamin D & COVID 19; Academic Censorship
- Coronavirus Pandemic Update 73: Relapse, Reinfections, & Re-Positives - The Likely Explanation
- Coronavirus Pandemic Update 72: Dentists; Diabetes; Sensitivity of COVID-19 Antibody Tests:
- Coronavirus Pandemic Update 71: New Data on Adding Zinc to Hydroxychloroquine +
- Coronavirus Pandemic Update 70: Glutathione Deficiency, Oxidative Stress, and COVID 19
- Coronavirus Pandemic Update 69: NAC Supplementation and COVID-19 (N-Acetylcysteine)
- Coronavirus Pandemic Update 68: Kawasaki Disease; Minority Groups & COVID-19:
- Coronavirus Pandemic Update 67: COVID-19 Blood Clots - Race, Blood Types, & Von Willebrand Factor
- Coronavirus Pandemic Update 66: ACE-Inhibitors and ARBs - Hypertension Medications with COVID-19
- Coronavirus Pandemic Update 65: COVID-19 and Oxidative Stress (Prevention & Risk Factors)
- Coronavirus Pandemic Update 64: Remdesivir COVID-19 Treatment Update
- Coronavirus Pandemic Update 63: Is COVID-19 a Disease of the Endothelium (Blood Vessels and Clots)?
- Coronavirus Pandemic Update 62: Treatment with Famotidine (Pepcid)?
- Coronavirus Pandemic Update 61: Blood Clots & Strokes in COVID-19; ACE-2 Receptor; Oxidative Stress
- Coronavirus Pandemic Update 60: Hydroxychloroquine Update; NYC Data; How Widespread is COVID-19?
- Coronavirus Pandemic Update 59: Dr. Seheult's Daily Regimen (Vitamin D, C, Zinc, Quercetin, NAC)
- Coronavirus Pandemic Update 58: Testing; Causes of Hypoxemia in COVID 19 (V/Q vs Shunt vs Diffusion)
- Coronavirus Pandemic Update 57: Remdesivir Treatment Update and Can Far-UVC Disinfect Public Spaces?

All coronavirus updates are at (including a discussion of that data for coronavirus outbreak in the UK and Brazil, coronavirus NYC, COVID 19 testing and infections, and other coronavirus news) and we offer many other medical topics (ECG Interpretation, strokes, thrombosis, pulmonary embolism, myocardial infarction, hypercoagulation, hypertension, anticoagulation, DKA, acute kidney injury, influenza, measles, mechanical ventilation, etc.).


Speaker: Roger Seheult, MD
Board Certified in Internal Medicine, Pulmonary Disease, Critical Care, and Sleep Medicine.

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Italian doctor on treating COVID-19 patients

Infectious disease specialist Dr. Giovanni Guaraldi discusses lessons being learned by Italian doctors about the new virus – with advice for Canadian physicians.

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Sounds of Coronavirus (COVID-19) - Lung Sounds

Coronaviruses are important human and animal pathogens. At the end of 2019, a novel coronavirus was identified as the cause of a cluster of pneumonia cases in Wuhan, a city in the Hubei Province of China. It rapidly spread, resulting in an epidemic throughout China, followed by an increasing number of cases in other countries throughout the world. It was declared a pandemic on March 11, 2020. It causes the respiratory disease COVID-19.



Coronavirus Signs & Symptoms:
Sounds of Asthma:
Sounds of Croup:






The content in this video is intended for educational purposes only. This video is intended to be viewed by medical professionals and healthcare providers. The content of this video is not meant to change, advise or direct any medical decision making. If you have any concerns you should always speak with your doctor or another healthcare provider. The graphical representations and sounds in this video are artistic renditions and simulations of pathology and do not accurately represent anatomical/pathological medical depictions.

The TRUTH of How Coronavirus Spreads and Coronavirus Prevention | Airborne Transmission

The TRUTH of How Coronavirus Spreads and Coronavirus Prevention | Airborne Transmission
#coronavirus #covid19

Coronavirus | COVID-19 YouTube Video Playlist:

Coronavirus Update : Coronavirus Transmission Through Air
It’s becoming clearer and clearer now, that this coronavirus spreads not just through contact and respiratory droplets that fly through the air like ballistics, but also it's being transmitted through the airborne route, meaning through aerosol, meaning the virus lingers in the air, and then someone inhales the virus. This is known as airborne transmission.

Let’s face it, there is a reason why hospitals with designated COVID-19 areas require everyone to wear an N95 respirator mask, as well as eye goggles. That’s because we know that this virus has the potential for airborne transmission. During normal breathing and speech, tiny particles are emitted mainly from the mouth. These particles can range in size, with the smallest being less than a micron (1 um), and the biggest being over 500 um in diameter.

To put some perspective on that, the average diameter of human hair is about 80 microns). Typically droplets that are less than 5 um are considered small, and its these small droplets that can be suspended in the air. Droplets that are over 100 um are considered large, and between 5 and 100 microns is intermediate. But the reality is, it’s a range of sizes, it’s a continuum, from less than 1 um to over 500 um. And more and more particles are emitted when someone is breathing heavier, such as with exercise, …..or if someone is coughing or sneezing, or if someone is shouting or singing.

Due to gravitational forces, particles that are bigger than 5 microns tend to settle, meaning fall down on surfaces such as the floor, and they fall fairly close to the source, typically within 6 feet. This is why the CDC recommends 6 feet for social distancing. But here’s the thing, sometimes these larger particles travel further than that, especially if someone is breathing heavy, or shouting, or singing, or coughing, or sneezing. Typically they fly no further than 12 feet in these situations. But we’re also spraying particles that are smaller than 5 microns, and its tiny particles that don’t act like ballistics, they act more like a gas cloud, where they float in the air, and travel up to 27 feet. The ones that are less than 1 um evaporate within milliseconds of hitting the air, while the particles that are more than 100 um can take up to a minute to evaporate.

What happens when the droplets that are less than 5 microns, what if they are spewed from someone who is infected with the virus, and all of a sudden in midair, they evaporate? Well, they dry out, and you’re left with a virus that is floating in the air. These are called droplet nuclei, aka aerosols. There are lots of factors that determine how long aerosols remain in the air. It depends on the person who emitted the particles, how they emitted them, the temperature, and humidity of the environment. Lack of airflow means this cloud will persist longer. And when this moist cloud finally does dissipate, you’re still going to have droplet nuclei that stay airborne….for about 3 hours, based on that NIH study.
At this point, we might not have 100% conclusive evidence that proves airborne transmission, but there are now several studies that strongly suggest that to be the case. Now just because we know that this virus spreads through the airborne route, that’s not to say that it doesn’t spread through contact and respiratory droplets, meaning the bigger droplets that act like ballistics. It spreads by all 3 of these mechanisms.

So handwashing is still important. As is not touching your face or mask with dirty hands. And maintaining 6 feet apart is a good thing, but its not good enough for certain situations. Remember earlier how I said when someone sneezes, that moist cloud containing aerosols can travel up to 27 feet?, And the virus can linger in the air for 3 hours. Some rooms have adequate ventilation that supplies clean outdoor air and minimizes recirculated air. The better the ventilation, the less likely the spread of aerosols. And even cracking open a window can make a huge difference, and having a fan blowing is good too. Other measures can help too, like having an air purifier with high-efficiency air filtration, and germicidal UV lights.

Dr. Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
Instagram Account:

#coronavirus #covid19

How Coronavirus Kills: Acute Respiratory Distress Syndrome (ARDS) & COVID 19 Treatment

How COVID-19 causes fatalities from acute respiratory distress syndrome (ARDS) by pulmonologist and critical care specialist Dr. Seheult of
This video illustrates how viruses such as the novel coronavirus SARS-CoV-2 can cause pneumonia or widespread lung inflammation resulting in ARDS.
Includes evidenced-based ARDS treatment breakthrough strategies: Low tidal volume ventilation, paralysis, and prone positioning.

Note: this video was recorded on January 28, 2020, with the best information available. Acute respiratory distress is, of course, not the ONLY way COVID 19 causes fatalities (other causes include heart failure, thrombosis (stroke), etc.)


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Video produced by Kyle Allred

MedCram = More understanding in less time

Topics from our COVID-19 pandemic series include: coronavirus spread, How Hospitals & Clinics Can Prepare for COVID-19, The ACE-2 Receptor - The Doorway to COVID-19 (ACE Inhibitors & ARBs), Flatten The COVID-19 Curve, Social Distancing, New Outbreaks & Travel Restrictions, Possible COVID-19 Treatments, Italy Lockdown, Global Testing Remains Limited, Coronavirus Epidemic Update 32: Data from South Korea, Can Zinc Help Prevent corona virus? Mortality Rate, Cleaning Products, A More/Less Severe Virus Strain? More Global COVID-19 Outbreaks, Vitamin D May Aid Prevention, Acute respiratory distress syndrome (ARDS), Rapid antigen tests, mutations, COVID-19 in Iran & more. has medical education topics explained clearly including: Respiratory lectures such as Asthma and COPD. Renal lectures on Acute Renal Failure, Urinalysis, and The Adrenal Gland. Internal medicine videos on Oxygen Hemoglobin Dissociation Curve / Oxyhemoglobin Curve and Medical Acid Base. A growing library on critical care topics such as Shock, Diabetic Ketoacidosis (DKA), aortic stenosis, and Mechanical Ventilation. Cardiology videos on Hypertension, ECG / EKG Interpretation, and heart failure. VQ Mismatch and Hyponatremia lectures have been popular among medical students and physicians. The Pulmonary Function Tests (PFTs) videos, how coronavirus causes morbidity and mortality, and Ventilator-associated pneumonia lectures have been particularly popular with RTs. NPs and PAs have provided great feedback on Pneumonia Treatment and Liver Function Tests among many others. Mechanical ventilation for nursing and the emergency & critical care RN course is available at Dr. Jacquet teaches our EFAST exam tutorial, lung sonography & bedside ultrasound courses. Many nursing students have found the Asthma and shock lectures very helpful. We're starting a new course series on clinical ultrasound & ultrasound medical imaging in addition to other radiology lectures.

Recommended Audience - Clinicians and medical students including physicians (MD and DO), nurse practitioners (NPs) , physician assistants (PAs), nurses (RNs), respiratory therapists (RTs), EMT and paramedics, and other clinicians. Review and test prep for USMLE, MCAT, PANCE, NCLEX, NAPLEX, NBDE, RN, RT, MD, DO, PA, NP school and board examinations. Continuing Medical Education (CME), MOC Points, CEU / CEs for medical professionals.

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#coronavirus #COVID19 #ARDS

How Coronavirus Kills Some People But Not Others - I'm a Lung Doctor (MEDICAL TRUTH) | COVID

How Coronavirus Kills Some People But Not Others - I'm a Lung Doctor (MEDICAL TRUTH) |
#coronavirus #covid19 #covid

⏩ Timestamps, click to skip ahead!
00:00 - Introduction
00:44 - How COVID Kills Some People (Starting of the explanation)
17:00 - How do we get COVID Patients with ARDS Better?
17:55 - Why do some COVID Patients get ARDS and Why do some Die?
20:55 - What do we know about COVID 19?

Coronavirus | COVID 19 YouTube Video Playlist:

Welcome to another video, for those of you who don’t know me, I’m Doctor Mike Hansen, I am a real doctor who specializes in pulmonary medicine, critical care medicine, and internal medicine.

Verify my board certification status:

When I’m not working in the hospital or pulmonary clinic, I’m at home working on making these videos for you, to deliver you accurate medical expertise, to the best of my ability (especially during this coronavirus pandemic).

This coronavirus, we know, is mainly transmitted by respiratory droplets, and through contact, by getting into our mucosa, like our mouth, nose, and eyes. Although less common, it also can be transmitted through aerosol, meaning airborne. Most likely when you have people in an enclosed space, such as an elevator, and someone sneezes or coughs without covering their mouths, and someone else can inhale it in.

This coronavirus attaches to cells in our body by this ACE2 receptor. This ACE2 receptor is only located on certain cells in our body. It's on our tongue, in our nose, back of the throat, and in our lungs. Specifically, within the lungs, it's only located on our type II alveolar cells.

We know that ARDS develops in about 4 to 5% of COVID-19 patients. And of all the people who get Coronavirus, the mortality rate is around 1 to 2% or max 3%. So why do some COVID-19 patients get ARDS, and why do some die? There are different reasons, and let's talk about them. It could be one of these reasons, but more likely it’s a combination of these reasons.

1) The coronavirus only gains entry into our cells that express the ACE2 receptor. They are located in multiple sites. Besides being in the lung, they’re in your mouth, nose, throat, stomach, small intestine, colon, skin, lymph nodes, thymus, bone marrow, spleen, liver, kidney, brain, and testes.

2) It makes sense that if the virus only gets into your mouth or nose or throat, but not the lungs, that it would cause only cold-like symptoms. But if the virus gets all the way down into the alveoli of your lungs, that’s what's going to cause ARDS. And by the way, the ACE2 receptors in your gut probably explains why some patients get nausea, vomiting, and diarrhea.

3) The amount of virus that you get into your body likely determines how sick you get. This is what we call the viral load.

4) The inflammatory reaction that occurs with COVID-19 is extremely complicated with lots of different proteins and hormones and interleukins at play. But there are several known genetic polymorphisms of these proteins that likely make some people more prone to getting worse illnesses than others. A genetic polymorphism simply means a variation on a particular gene. For example, there are genetic polymorphisms for the ACE gene, as well as IL-6. Basically, a lot of it just comes down to our genes. And sex.

5) Because the 5th reason has to do with estrogen. Estrogen is known to inhibit the effects of IL-6, which plays a huge role in this cytokine storm. This might explain why women overall have less severe disease compared to men.
6) And the 6th reason is because of people who are already taking certain medications. For those people who are already on and ACEI such as lisinopril, or an ARB such as losartan, or telmisartan, or candesartan, or irbesartan. Or people who take hydroxychloroquine for lupus or rheumatoid disease. Or people who take tocilizumab, an IL-6 receptor inhibitor. Are these coronavirus patients less prone to getting severe illness? My guess is yes.

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Dr. Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
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Italians use creativity to counter damage from COVID-19

As Italy re-opens, businesses in the design and fashion capital of Milan see creativity as a way to counter the damage done by COVID-19.

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Lung Doctor Analyzes George Floyd Autopsy Report (MEDICAL EXPLANATION)

Lung Doctor Analyzes George Floyd Autopsy Report (MEDICAL EXPLANATION)

Let’s be clear..we’ve all seen the video by now. It's obvious that these police officers killed George Floyd. The Hennepin County Medical Examiner, and the independent medical examiner hired by the family of George Floyd, Dr. Michael Baden, have concluded that his death was a homicide….but their opinion differs on the cause of death. But if both of them declared that his death was a homicide, does the cause of death really matter? (YES). I want justice for George Floyd, and that is why I’m making this video, because the medical explanation for his cause of death, is not a simple explanation. As a lung doctor, part of my job is to figure out why people can’t breathe. As an intensive care doctor, part of my job is to care for people who are on the brink of death. Like when someone can’t breathe. So when someone dies of asphyxia, as is the case of George Floyd, the determination of the cause of death is dependent on information elicited based on the investigation, which includes, the deceased personal medical history namely, autopsy, and crime scene investigation, which of course includes video evidence. Asphyxia is a Greek term that translates to “loss of pulse.”

Mechanical asphyxia involves some physical force or physical abnormality that interferes with the uptake and/or delivery of oxygen. With asphyxia, the brain doesn’t get enough oxygen, and when the pons and the medulla aren’t getting enough oxygen, they can no longer function. This means they can no longer tell the diaphragm to contract, and breathing then stops. While this happens, the heart is also not getting enough oxygen, and typically the heart pumps slower and slower until it stops. Prolonged continuous application of extreme pressure on the thorax, such as with the bodyweight of several officers, is capable of causing death. This is important, because this contributed to the death of George Floyd, in addition to the knee to the neck. The neck contains our airway, the trachea, and it also contains carotid and vertebral arteries and jugular veins. The arteries here deliver oxygenated blood to the brain, while the jugular veins allow the deoxygenated blood to flow back to the heart. So what happens when pressure is placed on the neck? Well, it depends, on a lot of different factors (amount and duration of pressure, etc). And looking at the George Floyd video, he was unconscious for more than 2 minutes with the knee still on his neck. There’s no doubt, that during this time, he took his last breath, and right around the same time, lost his pulse. By the time the EMS guy checks his pulse, I highly doubt he actually felt a pulse, because it was more than two minutes after George lost consciousness. It was obvious that when they moved George onto the stretcher, he was completely limp because he was dead. And it wasn’t until much later, did they start CPR, in the ambulance. Now let’s get to what the medical examiners had to say about this case.

Dr. Michael Baden, who did the independent autopsy says Floyd died of asphyxiation from sustained pressure when his back and neck were compressed, with the neck pressure cutting off blood flow to his brain.” I agree with that assessment. I would also add that partial compression of the trachea, causing airway compromise, was also possible. The weight on George’s back made the work of breathing much harder for his diaphragm, and the neck pressure at the very least meant less blood (and thus oxygen) was being delivered to his brain, and less carbon dioxide could be removed from his brain. After a while, the diaphragm becomes fatigued, and no longer has the strength to contract, which means the lungs can’t get oxygen into the blood, and can’t get carbon dioxide out of the blood. And all of this caused him to lose consciousness. And probably within seconds, he lost a pulse. And despite losing consciousness, and despite losing a pulse, they continued to apply pressure on the neck, and put their weight on his back. The Hennepin County medical examiner's office said that the cause of death is cardiopulmonary arrest complicating law enforcement subdual, restraint, and neck compression. This statement doesn’t really make sense to me. But the Hennepin County release also says heart disease was an issue; the independent examiner didn't find that. The county said that fentanyl and methamphetamine use were among significant conditions, but its report didn't say how much of either drug was in Floyd's system or how that may have contributed. But Dr. Michael Baden got it right.

- Doctor Mike Hansen

VERIFY: Is coronavirus caused by a bacterial infection?

A Facebook post about a study in Italy has several viewers asking if it’s true that autopsies completed really found that COVID-19 is a bacteria, not a virus.



Anny trying to make USA more normal. Cooking Tangyuen. Found Coronavirus Patent Zero. Paranoia in China as people are going a little crazy. Meeting my family for my mom’s birthday. Eva makes some new friends.

Article about Patient Zero (translated with google translate)

[The first anatomy of the deceased person with new crown pneumonia performed: #The anatomy is expected to get a pathology report within 10 days #] at After the legal policy allowed and obtained the consent of the patient's family members, the autopsy work of the two patients who died of new pneumonia was completed on the 16th in Wuhan Jinyintan Hospital. Forensic pathologists who participated in the autopsy work. Pathology has been submitted for examination, and conclusions are expected within 10 days. Liu Liang introduced that through pathological anatomy and subsequent inspection, the distribution of the virus in the human body can be most intuitively observed under a microscope, which organs, tissues, and cells have suffered the most damage, and where are the weaknesses of the enemy, making it a clinician The diagnosis and treatment provide clues. Liu Liang also mentioned that the existence of fecal-oral transmission can also be found in the results of dissection and virus testing. Implementation of the Anatomy of the Death of the New Patient with New Coronary Pneumonia: Expected within 10 days ..

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FAQs about Autopsies: Do You Always Find a Cause of Death?

Dr Suzy Lishman, Consultant Histopathologist at Peterborough Hospital, goes through some of her commonly asked questions surrounding her Living Autopsy lecture.
Full timings below.
00:03 Do all pathologists perform autopsies?
01:10 How did you feel when you did your first post-mortem?
02:08 Can you donate your organs after an autopsy?
03:25 Do you always find a cause of death?
Watch the full lecture to find out more:
Thinking of a career in histopathology? Go to:
If you’re thinking of running your own Living Autopsy event, it’s worth noting how Dr Suzy Lishman answers each of these questions. The answers are simplified, they don’t use jargon and they’re answered with an enthusiasm for pathology as a whole, as well as just for histopathology.
Filming credit:
Editing credit: Rachel Berkoff

Corona deaths को Italy, postmortem, WHO, 5G radiation से जोड़ती conspiracy theory का Fact check

अगर आपको किसी ख़बर पर शक है, तो पड़ताल के लिए भेजें, हमारे वॉट्सऐप नंबर पर. क्लिक करें:

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स्टोरी पढ़ने के लिए यहां क्लिक करें:

A long-read text message is viral on social media platforms and in WhatsApp groups regarding 5G radiation theory in connection with Coronavirus Infection. Many users claimed that the new findings came out after Italy performed a post-mortem of a Corona patient for the first time in the world. The Lallantop investigated the various claims one by one and found the claims false and misleading. Coronavirus does not transmit through radio waves or mobile networks. Watch the fact check video in Hindi.

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Medical Expert on Autopsy Findings of COVID-19

Autopsies of 29 COVID-19 victims have found the virus could damage human lungs, bronchi and immune system, said a medical expert at a press conference in Beijing on Tuesday.

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COVID-19 : les résultats des deux premières autopsies seront soumis à un examen pathologique

Le 16 février à 3 heures du matin, la première autopsie en Chine d’un patient décédé du COVID-19 a pris fin à l'hôpital Jinyintan de Wuhan, avec le consentement de la famille du patient et sous réserve des lois et politiques du pays. La pathologie du COVID-19 a pu être identifiée avec succès. Le même jour, à 18 heures 45, la deuxième autopsie d’une autre patiente décédée de la même maladie a pu également être menée à bien dans cet hôpital. Les résultats obtenus au terme de ces deux autopsies vont être soumis à un examen pathologique.

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