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Diabetic bullae (Bullosis Diabeticorum) - Clinical essentials


Diabetic bullae (Bullosis Diabeticorum) - Clinical essentials

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Diabetic bullae (Bullosis Diabeticorum) - Clinical essentials

Bullous disease of diabetes (bullosis diabeticorum) is a distinct, spontaneous, noninflammatory, blistering condition of acral skin that is unique to patients with diabetes mellitus. Bullous disease of diabetes tends to arise in long-standing diabetes or in conjunction with multiple complications. The etiology of the disease is yet unknown. In the United States, bullous disease of diabetes has been reported to occur in approximately 0.5% of diabetic patients. Male patients have twice the risk as female patients.

Kramer first reported bullous-like lesions in diabetic patients in 1930 ; Rocca and Pereyra first characterized this as a phlyctenar (appearing like a burn-induced blister) in 1963. Cantwell and Martz are credited with naming the condition bullosis diabeticorum in 1967. It is also termed bullous disease of diabetes and diabetic bullae.

While lesions typically heal spontaneously within 2-6 weeks, they often recur in the same or different locations. Secondary infections may also develop; these are characterized by cloudy blister fluid and require a culture.

The clinician should consider direct immunofluorescence (DIF) studies to exclude histologically similar entities (eg, noninflammatory bullous pemphigoid, epidermolysis bullosa acquisita, porphyria cutanea tarda, other bullous porphyrias), as DIF studies are only rarely positive in bullosis diabeticorum.

Pseudoporphyria blistering due to photosensitizing drugs, chronic dialysis regimens, or ultraviolet A tanning devices should also be considered.

Specific treatment is unwarranted unless secondary infections (eg, staphylococcal) occur, thereby warranting antibiotic therapy. However, aspiration of fluid from lesions using a small-bore needle might help prevent accidental rupture.

#Diabeticbullae #Bullosisdiabeticorum #Clinicalessentials #clinicalskills #diabetes #diabeticcomplications #diabetesrisks #medicine #mbbs #internalmedicine #usmle #neetpg #fmge #usmlestep2ck #usmlestep1 #plab

Intersection syndrome - Clinical essentials

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Intersection syndrome - Clinical essentials

Intersection syndrome is a condition that affects the first and second compartments of the dorsal wrist extensors. The condition is thought to occur as a result of repetitive friction at the junction in which the tendons of the first dorsal compartment cross over the second, creating tenosynovitis. This is typically noted as a pain just proximal and dorsal to the radial styloid, or also noted anatomically by 4 cm - 6 cm proximal to Lister's tubercle.

The term intersection syndrome refers to the intersection (at an angle of around 60°) of the musculotendinous junctions of the first and second extensor compartment tendons.

The intersection syndrome is a relatively uncommon disorder that is often misdiagnosed with other conditions, such as ‘De Quervain’s tenosynovitis.

#intersectionsyndrome #clinicalskills #clinicalessentials

McMurray’s Test for Meniscal Tear : Clinical essentials

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McMurray’s Test for Meniscal Tear

Commonly used test in orthopaedic examination to test for tear of the meniscus .This test is a rotational maneuver of the knee that is frequently used in the examination of the patient in the diagnosis of meniscal tears. Meniscal tears are very common .When patient sustains an injury of knee and has a meniscal tear, usually the patient complaints of knee pain localized to the medial or lateral side of the knee.The patient also have locking and clicking .Sometimes patient will have an effusion

• Joint line tenderness is the most sensitive finding .It can be on the medial side (medial meniscal tear ),or on the lateral side (lateral meniscal tear).
• Minimal swelling of the knee.
• Possible extension lag (locked knee),due to displaced bucket handle tear of the meniscus.
• Pain at higher level than the joint is usually associated with medial collateral ligament tear.pain at the lower level is usually associated with the pes anserine bursitis.

#mcmurraystest #meniscaltear #physiotherapy #physicaltherapy #clinicallearning #clinicalessentials #McMurrayTest

Classic bullous pemphigoid (Clinical essentials): Dr. Aashritha yerneni

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Classic bullous pemphigoid (Clinical essentials): Dr. Aashritha yerneni

Pathogenesis includes development of autoantibodies against hemidesmosome adhesion complex in the basement membrane of the skin (BP180 and/or BP230), resulting in complement activation, mast cell degranulation, release of inflammatory mediators, and recruitment of neutrophils and eosinophils. This leads to the formation of localized or generalized urticarial plaques, erythematous papules or plaques, and/or bullae. The disease can occur on any body surface, but mucous membrane involvement is seen in only 10%-30% of patients.

BP has variable clinical presentation as well as disease course. Presentation can range from itch without rash to localized or generalized eruptions. The morphology may be variable: some presentations are nonbullous, in which case urticarial or eczematous-appearing papules and plaques, or excoriations only, may be seen. The localized variant can mimic dyshidrosis when on the palms or soles and can develop in sites of radiation, surgery, prior photodynamic therapy, infection, paralysis, and venous stasis, as well as adjacent to ostomies. Disease course ranges from self-limiting to chronic over months to years.

Peripheral eosinophilia can be seen in approximately 50% of patients and is positively correlated with disease severity. Its presence has also been correlated with older age and palmoplantar involvement. Lack of peripheral eosinophilia has been positively correlated with younger age and mucosal disease.

BP is associated with neurologic disease ranging from stroke to dementia. Relapse of BP has been shown to be more likely in individuals with extensive disease and dementia. BP is also been associated with other autoimmune diseases such as diabetes mellitus, thyroiditis, dermatomyositis, systemic lupus erythematosus, rheumatoid arthritis, ulcerative colitis, myasthenia gravis, and multiple sclerosis in case reports and case series. A population-based study from Finland strongly suggests an increased risk of BP in patients with dermatitis herpetiformis. Medications (ie, furosemide, NSAIDs, captopril, penicillamine, gliptins, and some antibiotics) have also been associated with triggering BP.

#Classicbullouspemphigoid #bullouspemphigoid #pemphigoid #clinicalessentials #dermatology #clinicalskills #clinicalvideos #usmlestep2ck #usmlestep2cs #usmle #neetpg

Diabetes Blisters & Rubbing Solved Using Dr Comfort Shape to Fit Socks - Leonard Genton Podiatry

Samantha explains why you should wear the Dr Comfort Shape to Fit socks if you have diabetes.

Heliotrope rash in Dermatomyositis - Clinical Essentials

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Heliotrope rash is caused by dermatomyositis (DM), a rare connective tissue disease. People with this disease have a violet or bluish-purple rash that develops on areas of the skin. They can also experience muscle weakness, fever, and joint aches.

The rash may be itchy or cause a burning sensation. It commonly appears on sun-exposed areas of the skin, including the:

face (including eyelids)
It isn’t uncommon for a person with this condition to have purple eyelids. The purple pattern on the eyelids may resemble a heliotropeflower, which has small purple petals.

DM is rare. In the United States, researchers believe there are up to 10 cases per 1 million adults. Likewise, there are about three cases per 1 million children. Women are more commonly affected than men, and African-Americans are more commonly affected than Caucasians.

What causes heliotrope rash?
The rash is a complication of DM. This connective tissue disorder has no known cause. Researchers are trying to understand who is likely to develop the disorder and what increases their risk.

Possible causes of dermatomyositis include:

Family or genetic history: If someone in your family has the disease, your risk may be higher.
An autoimmune disease: A functioning immune system attacks unhealthy or invading bacteria. In some people, however, the immune system attacks healthy cells. When this happens, the body responds by causing unexplained symptoms.
Underlying cancer: People with DM are at a higher risk for developing cancer, so researchers are investigating whether cancer genes play a role in who develops the disorder.
Infection or exposure: It’s possible that exposure to a toxin or trigger could play a role in who develops DM and who doesn’t. Likewise, a previous infection may also affect your risk.
Complication of medication: Side effects from some medications could lead to a rare complication like DM.

#heliotroperash #dermatomyositis #clinicalessentials #clinicalskills #clinicals #usmlestep1 #usmle #mbbs #mbbs #nationalexittest #next1 #next2 #usmlestep2ck

Diabetic Dermopathy || Shin spots

This is a medical educational video on Diabetic Dermopathy or Shin spots!

Crepitus of the Joints - Clinical essentials

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Crepitus of the Joints - Clinical essentials

Crepitus is the abnormal popping or crackling sound in either a joint or the lungs, which may be faint or loud enough for people to hear. It is often accompanied by a popping or crunching sensation that may sometimes be uncomfortable or painful.

Crepitus in the joints is typically related to joint damage. Crepitus in the lungs is caused when collapsed or fluid-filled air sacs abruptly open upon inspiration.

Crepitus of the Joints
Crepitus may occur in tandem with a joint disorder or entirely on its own. As a symptom, it is not inherently problematic. Cracking your knuckles, for example, is a form of crepitus wherein tiny nitrogen bubbles in a joint suddenly pop with strenuous movement.

It is generally only a problem when the popping is progressive or is accompanied by symptoms of joint damage, injury, or infection.

Joint Damage
Crepitus can occur when the roughened surfaces of two joints rub together, causing the physical grating of cartilage and/or bone.

If the pain is felt, it is typically related to advanced joint damage and/or the compression of nerves between narrowed joint spaces. It is at this stage that the joint may begin show signs of injury, including swelling, redness, reduced range of movement, and malformation.

Osteoarthritis (wear-and-tear arthritis) is the most common cause of this, although crepitus can occur with other forms of arthritis, including rheumatoid arthritis, psoriatic arthritis, gout, and juvenile idiopathic arthritis. It can affect any joint of the body but is most common in the knees, hands, feet, lower back, hips, and shoulders.

#crepitus #crepitusoftheknee #crepitusofthejoints #whatiscrepitus #crepitusmechanism #clinicalessentials #clinicalskills #orthopedics #orthopeadics #usmle #usmlestep1 #mbbs #clinicalvideos #nationalexittest #nationalexitexam

Necrobiosis lipoidica diabeticorum / ডায়াবেটিস রোগীর চর্মরোগ

Necrobiosis lipoidica diabeticorum হলো একটি বিশেষ ধরনের চর্মরোগ, যা দীর্ঘমেয়াদে ডায়াবেটিস রোগীর ত্বকে হতে পারে।

ডায়াবেটিস রোগীর হাইপোগ্লাইসেমিয়া কেন হয়? Causes of hypoglycemia in diabetes patient.


Bouchard’s and Heberden’s nodes : Clinical essentials

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Bouchard’s and Heberden’s nodes

A Heberden node describes a bony swelling of the distal interphalangeal finger joint. It is a sign of osteoarthritis, a degenerative joint disease.
A Bouchard node is a similar swelling affecting the proximal interphalangeal finger joint .

Who gets Heberden and Bouchard nodes?

Heberden and Bouchard nodes are equally common in males and females of all races.
They are common in older individuals. However, more than half of the patients with Heberden nodes and osteoarthritis are diagnosed before the age of 65 years.
Bouchard nodes are less common than Heberden nodes and are associated with more severe arthritis.
The presence of the nodes is strongly familial.
More than 60% of patients with osteoarthritis of the knees have Heberden nodes.

What causes Heberden and Bouchard nodes?

A Heberden or Bouchard node is due to an exostosis — a bony enlargement — and a sign of osteoarthritis. The cause of osteoarthritis is not fully understood. There is a genetic predisposition to the development of nodes .
Osteoarthritis is considered a sign of 'wear and tear' in a joint. The earliest structural abnormalities in osteoarthritis are evident in the ligaments supporting the joints . The cartilage of the joints breaks down, osteophytes (bone spurs) develop, and the ends of the bones rub against each other.

Inflammation in the nodes is due to friction-induced capsular rupture and synovial leakage .

What are the clinical features of Heberden and Bouchard nodes?
A Heberden node is a bony swelling of a distal interphalangeal joint and a Bouchard node is a bony swelling of the proximal interphalangeal joint.

Bony swelling can affect either the lateral or midline aspects of the joint or both .
Multiple nodes can be present on one digit.
The nodes can affect one or many digits.
Nodes most often affect the middle finger or thumb of both hands.
They may grow slowly or rapidly.
The nodes may become inflamed and painful but are often painless and unnoticed.
A Heberden node may be associated with a digital myxoid pseudocyst.
As well as affecting fingers, osteoarthritis causes discomfort, pain, and stiffness in other joints of the hands, knees, hips, and spine.

How are Heberden and Bouchard nodes diagnosed?
The diagnosis of Heberden or Bouchard node is usually made clinically due to their characteristic appearance. Imaging may be performed on the affected digit.

Ultrasound scanning reveals osteophytes, synovitis, and bony erosions of osteoarthritis.
A plain X-ray of the affected joint may show interphalangeal osteophytes, although correlation is poor with clinical Heberden nodes. Osteophytes are more common in patients with inflammatory arthritis.
Magnetic resonance imaging (MRI) may confirm the diagnosis of arthritis in the affected digit. Heberden and Bouchard node formation is associated with soft tissue bulging through the capsule between the dorsal tendons and collateral ligaments. Ligamentous change can be seen in adjacent but clinically normal joints.
Histological examination of Heberden or Bouchard node is not commonly undertaken. It may show the presence of osteophytes and dorsal contractures.

How are Heberden and Bouchard nodes treated?

There is no specific treatment to repair a Heberden or Bouchard node.
Treatment for osteoarthritis may include lifestyle changes such as exercise, weight loss, low-inflammatory diet, heat bags, and cold compresses.
Medical treatments include pain relief and nonsteroidal anti-inflammatory drugs .
Some patients with osteoarthritis may require surgery to repair or replace one or more joints.

#bouchardnodes #heberdennodes #dermatology #clinicalessentials #neetpg #usmle #mbbs #clinicalskills #medvizz

Dermatology: (Wk#1)(Wed)(Spr-20): Bullous pemphigoid, Psoriasis, and histology review of the skin.

In this video Dr. Gillard recaps the pertinent Histology of the skin. He also goes over to dermatological conditions: bullous pemphigoid, and psoriasis.

Granulomatous Skin Diseases 101: Dermpath Basics for Dermatology Pathology & USMLE

A recording of a rapid fire fundamental level dermpath session for my awesome dermatology and pathology residents. Slide set courtesy of my colleague and friend, Dr. Tammie Ferringer.

List of entities discussed (click timestamp to go to that part of video):

“Wolverine sign”: Clue for Foreign body granuloma (silica in this case) 0:00
Coccidiodomycosis (Coccidioides immitis) with pseudoepitheliomatous hyperplasia 3:00
Necrobiotic xanthogranuloma (NXG) 7:20
Eruptive xanthoma (associated with hypertriglyceridemia) 11:50
Langerhans cell histiocytosis (LCH) 14:00
Epithelioid sarcoma (mimic of rheumatoid nodule & deep granuloma annular) 17:03
Rosai Dorfman disease 20:25
Xanthelasma 24:25
Tips for recognizing eyelid skin histology 25:00
Necrobiosis lipoidica (diabeticorum) (NLD) 26:28
Juvenile xanthogranuloma (JXG) aka solitary xanthogranuloma 29:00
Granuloma annulare 32:13
Granuloma annulare, interstitial pattern 35:43
Reticulohistiocytoma (Solitary Epithelioid Histiocytoma) & Reticulohistiocytosis 38:19
Sarcoidosis 41:33
Rheumatoid nodule 43:02
Vs deep granuloma annulare 45:35
Mucocele of Lip (mucous extravasation phenomenon) 47:00
Chalazion (ruptured inflamed Meibomian gland in eyelid) – not a stye (hordeolum) 49:18
Cookie Monster shaped Meibomian gland 49:40
Cutaneous Crohn disease (inflammatory bowel disease) in perianal skin polyp 51:24
Verruciform xanthoma 53:40
Lupus miliaris disseminatus faciei (FIGURE, acne agminata, granulomatous rosacea) 56:08
Ruptured cyst with keratin granuloma 57:55
Suture granuloma with scar 58:45
Granulomatous tattoo reaction vs sarcoidosis in a tattoo via koebnerization/Koebner phenomenon 1:00:28
Gel foam under the microscope (in a skin punch biopsy site) 1:02:40
Tuberous xanthoma (associated with familial dysbetalipoproteinemia III & other serum lipid abnormalities) 1:03:37

- WSI digital slide (metastatic breast carcinoma mimicking GA):
- WSI digital slide of classic rheumatoid nodule:
- Tendon vs Nerve Histology Made Simple with the Ramen Noodle Sign (of Fulton) video:

Interstitial GA-like pattern can be seen in extra-genital lichen sclerosus. Here's our paper about this:

A complete organized library of all my videos, digital slides, pics, & sample pathology reports is available here: (dermpath) & (bone/soft tissue sarcoma pathology).

Please check out my Soft Tissue Pathology & Dermatopathology survival guide textbooks: ‬

This video is geared towards medical students, pathology or dermatology residents, or practicing pathologists or dermatologists. Of course, this video is for educational purposes only and is not formal medical advice or consultation.

Presented by Jerad M. Gardner, MD. Please subscribe to my channel to be notified of new pathology teaching videos.

Follow me on:
Snapchat: JMGardnerMD
Twitter: @JMGardnerMD
Instagram: @JMGardnerMD

Acidity (Clinical essentials) - Dr. Kiran Peddi MRCP(UK), FRCP(London), CCT(Gastro)

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What is acidity?
The food we eat goes into our stomach through the oesophagus. The gastric glands in your stomach create acid, which is necessary to digest the food. When the gastric glands create more acid than needed for the digestion process, you tend to feel a burning sensation below the breastbone. This condition is commonly called acidity.

Acidity, also called acid reflux, is a condition that is characterised by heartburn that is felt around the lower chest area. It is a common condition that occurs when stomach acid flows back up into the food pipe. The most common acid reflux symptom is a burning sensation in the chest, and pain. While most people suffer in pain, they do not realise that poor lifestyle choices are the main reason behind acidity.

When acidity symptoms occur more than twice a week, your doctor may diagnose you with Gastroesophageal reflux disease or GERD. Chronic acidity may lead to serious risks that include:

Oesophagus damage: Oesophagus is the tube that connects your mouth to your stomach. When acid moves back up and enters the oesophagus, it sets the stage for esophageal ulcers, oesophagitis, esophageal strictures, and Barrett’s Oesophagus.
Increased risk of oesophagus cancer, which is more likely if you have a history of the condition in the family.
Acid reflux can wear down the enamel of your teeth and lead to cavities.

Acidity symptoms
Acidity symptoms differ from one person to another. Most things that contribute to gas also lead to acidity meaning, gas and acidity symptoms are almost similar. While the most common acid reflux symptoms are chest pain and burning sensation below the breastbone, there are other symptoms that are uncommon. Acidity symptoms include the following:

Burning sensation and pain in the stomach
Burning sensation and pain in the throat
Difficulty swallowing or the sensation of food being stuck in your throat
Frequent burping or hiccups for no apparent reason
Burning sensation and pain in the chest
Regurgitation: Prolonged sour taste in the mouth or bitter-tasting acid that backs up into your throat and mouth
Post-meal heaviness
Bad breath

Acidity treatment
When you visit your healthcare provider, your symptoms are considered to determine whether you have acidity or whether it is a possible complication of some other problem. Based on your condition and the depth of the acid reflux problem,

You will be prescribed an antacid that contains aluminium, calcium or magnesium.
Your doctor may also suggest the use of histamine blocking agents (H2 receptor blockers) such as cimetidine, nizatidine, ranitidine, and famotidine
If the condition is severe, proton pump inhibitors may be prescribed
If the condition is extremely severe, your doctor may suggest Vagotomy surgery that helps to reduce the production of acid in the stomach.
Your healthcare provider may be able to suggest certain do’s and don’ts based on your health, which will help you control acidity.

Home remedies for acidity
Acidity is a common problem that most of us face at least once in our life. Most of us prefer home remedies over medications. Some of the home remedies for acid reflux include the following.

Coconut water: This tasty treat is known to soothe your stomach and the digestive system. Take at least two glasses a day.
Watermelon juice: It is great to counter acidity. You can take a glass of watermelon juice with breakfast.
Fresh lime juice that is taken at least an hour before lunch helps to reduce the uneasiness caused by acidity.
A glass of buttermilk after a spicy meal helps reduce the uneasiness as buttermilk contains lactic acid that normalises acidity in the stomach
You can either munch on a few basil leaves, or boil them in water and drink it frequently to reduce acid reflux. You can also do this with mint leaves.
Drink a glass of lukewarm water after every meal
Include banana, cucumber and yoghurt in your diet. They are known to give instant relief from acidity.
Surprisingly, sucking on a piece of clove when you have acidity helps to reduce the symptoms.
Ginger aids in digestion. Use ginger in your cooking or boil it in a glass of water, reduce to half glass and consume the water.
Cumin is a great remedy for acidity. Munch on some cumin or boil a teaspoon of cumin in a glass of water. Boil until the water is reduced to half. Drink it on an empty stomach.
Try chewing gum! As unbelievable as it may sound, chewing gum generates saliva that helps to move the food through the oesophagus.
Take a tablespoon of apple cider vinegar with a glass of water every morning on an empty stomach.
Drink at least two litres of water every day.

Psoriasis And Diabetes: What Is The Link?

Psoriasis And Diabetes: What Is The Link?

Disclaimer: The materials and the information contained on this channel are provided for general and educational purposes only and do not constitute any legal, medical or other professional advice on any subject matter. Always seek the advice of your physician or other qualified health provider prior to starting any new diet or treatment and with any questions you may have regarding a medical condition. If you have or suspect that you have a medical problem, promptly contact your health care provider.

Asthma Attacks: Causes And Symptoms:

Gastrointestinal Bleeding: Causes And Symptoms:

High Cholesterol And Weight: Causes And Risk Factors:

Is It Possible To Lower Cholesterol Quickly?:

Diabetic Dermopathy: Causes And Prevention:

Elbow Pain When Lifting: Causes And Treatment:

COVID-19 And Black Fungus: What Is The Link?:

Pain In The Middle Of The Bicep: Main Causes:

Pulmonary Arterial Hypertension (PAH): Causes And Symptoms:

Heart Murmurs: Causes And Symptoms:

Black Fungus And COVID-19: Myths And Facts:

Circulatory System Diseases: Can They Be Prevented?:

Left Atrial Enlargement: Causes And Symptoms:

Blurry Vision In One Eye: Is It Serious?:

Morbid Obesity: Symptoms And Prevention:

Giant Cell Myocarditis: Causes And Symptoms:

Mitral Valve Prolapse: Causes And Symptoms:

Atherosclerosis (Arteriosclerosis): Symptoms And Treatment:

#diabetesandpsoriasis #healthyfit

Massage Therapy Study Guide 19:. Condition Treatment Plan: DIABETES MELLITUS


Necrobiosis lipoidica تضرر الطبقة الشحمية للجلد (أو البلى الفيزيولوجي الشحماني السكري)

‏Necrobiosis lipoidica
هو اضطراب تنكس الكولاجين مع استجابة حبيبية ، سماكة جدران الأوعية الدموية ، وترسب الدهون. المضاعفات الرئيسية للمرض هي التقرح ، وعادة ما يحدث بعد الصدمة. يمكن أن تحدث العدوى ولكنها غير شائعة. تم الإبلاغ عن حالات نادرة من سرطان الخلايا الحرشفية التي تتطور في الآفات المزمنة للنخر الشحمي.

#Necrobiosis #lipoidica is a disorder of collagen degeneration with a granulomatous response, thickening of blood vessel walls, and fat deposition. The main complication of the disease is ulceration, usually occurring after trauma. Infections can occur but are uncommon. There have been rare reported cases of squamous cell carcinomas developing in chronic lesions of necrobiosis lipoidica.

تضرر الطبقة الشحمية للجلد (أو البلى الفيزيولوجي الشحماني السكري) (Necrobiosis lipoidica diabeticorum)
إن ضعف إمدادات الدم للجلد (والتي تحدث نتيجة الإصابة بمرض السكري لفترة طويلة دون مراقبة جيدة لنسبة السكر في الدم)، مما يسبب تغيرات في الكولاجين والدهون الموجودة تحت الجلد، بحيث يصبح الجلد رقيقا وذا لون أحمر، وغالباً ما تصيب هذه الآفات الأجزاء السفلية من الساقين، ويمكن أن تتحول إلى قرحة إذا تعرض المكان لأذى خارجي.

تبدو الآفات من هذا النوع محددة الحدود، وفي بعض الأحيان يمكن أن يشعر المريض بالحكة والألم، وهذه الحالة لا تحتاج إلى علاج طالما أن الآفات لم تفتح (أي أن الطبقة الخارجية من الجلد لا تزال سليمة)، إلا أنها تستوجب الذهاب للطبيب لتلقي العلاج عندما تفتح هذه الآفات.

كذلك فإن المريض في كلتا الحالتين يحتاج إلى تنظيم ومراقبة مستوى السكر في دمه.

What Causes Diabetic Bullae? |శరీరం పై నీటి బొబ్బలకు కారణం | Dr.ETV | 22nd February 2021

#WhatCausesDiabeticBullae #DrETV#Health #ETVWin

శరీరం పై నీటి బొబ్బలకు కారణం

Dermatologist Dr Swapna Priya addresses the viewer's query on the causes of diabetic bullae and briefs the treatment methods.

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Inflammatory Dermpath 101 (A Beginner's Guide to Diagnosing Skin Rashes for Non-Dermatopathologists)

A basic algorithmic approach to the microscopic diagnoses of inflammatory skin disease aimed at medical students, residents, general surgical pathologists, and other non-dermatopathologists. Presented virtually as the 2021 Bouldin Lectureship Grand Rounds lecture at the University of North Carolina Department of Pathology & Laboratory Medicine, April 8, 2021. All images in the video are from my Survival Guide to Dermatopathology textbook (available here: ‬

A complete organized library of all my videos, digital slides, pics, & sample pathology reports is available here: (dermpath) & (bone/soft tissue sarcoma pathology).

Topics discussed and timestamps:
***HUGE thanks to viewer Tonia Syrnioti for typing these up for me! Thank you!!!***

Intro 0:00
How to approach an inflammatory skin biopsy 5:56
Spongiotic pattern 9:16
List of diseases 11:04
Presence of eosinophils 11:46
Langerhans microabscesses 12:46
Subcorneal pustule 13:58 (spongiosis + eosinophils + subcorneal pustule: AGEP)
Do a PAS stain - think of dermatophyte infection
Scale (parakeratosis) crust (pink serum) 16:10
Psoriasiform pattern 16:58
List of diseases 19:20
Munro's microabscess 19:58
Guttate psoriasis 20:08
Psoriasiform + lichenoid + plasma cells - Do spirochete stain for secondary syphilis!
Vacuolar & Lichenoid interface pattern 20:40
Vacuolar interface pattern 20:48
List of diseases 22:38
Lichenoid interface pattern 23:38
List of diseases 23:52
Lichen planus 24:48
Hypertrophic lichen planus 26:50
Lichen sclerosus et atrophicus 28:25
Lichen simplex chronicus 32:46
Prurigo nodularis 34:36
Stevens- Johnson's syndrome/TEN 36:08 (can look identical to Erythema multiforme)
Lupus erythematosus 37:26
Discoid lupus erythematosus 38:06 (can look like AK/SCC)
Superficial perivascular pattern 38:49
List of diseases 38:58
Urticaria 39:08
Superficial & deep perivascular pattern 39:21
List of diseases 39:39
Arthropod bite reaction 40:01 (can look like bullous pemphigoid)
Wells syndrome 40:29
Perniosis (chilblains) 41:02
Granulomatous pattern 41:19
List of diseases 41:45
Remember to look for fungi & foreign material
Sarcoidosis - diagnosis of exclusion
Palisading necrobiotic granulomatous pattern 42:15
List of diseases 42:49
Granuloma annulare 43:25
Elastophagocytosis 43:35
Rheumatoid nodule 43:55
Mimic: epithelioid sarcoma 44:45
Necrobiosis lipoidica 45:38
Necrobiotic xanthogranuloma 46:00
Leukocytoclastic vasculitis pattern 46:22
List of diseases 47:58
Polyarteritis nodosa 48:05
Thrombotic vasculopathy pattern 48:34
List of diseases (often medical emergencies!!) 49:20

This video is geared towards medical students, pathology or dermatology residents, or practicing pathologists or dermatologists. Of course, this video is for educational purposes only and is not formal medical advice or consultation.

Presented by Jerad M. Gardner, MD. Please subscribe to my channel to be notified of new pathology teaching videos.

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Diabetic Neuropathy

What Kind of Skin Problems Do People with Diabetes Get | diabetes,skin condition | Dr Vrinda Agrawal

Watch► What Kind of Skin Problems Do People with Diabetes Get| diabetes, skin condition | Dr Vrinda Agrawal

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